👉 Ready to level up your life? 🚀 Discover my 10 Hacks to Improve Your Life & Longevity Playbook! Smarter, healthier, and more exciting living starts here. Grab your free copy now at longevity-and-lifestyle.com/freebie! Brain health isn't just about the occasional crossword puzzle or choosing that salad over the cheeseburger; it's a complex interplay between stress, sleep, attention, and much more. Dive deep into the cerebral world with cognitive neuroscience maven, Dr. Andrew Hill. Not only does Dr. Hill lead the charge at the Peak Brain Institute, but his background in neurofeedback and brain optimization is nothing short of revolutionary. Join us as we explore the transformative power of brain training, the nuances of neurofeedback, and the potential for rewiring our mental processes to combat age-related decline. If that’s not enough, stay tuned for a brain mapping walkthrough that demystifies EEG analysis and lays the groundwork for personalized cognitive enhancement protocols. From the intricacies of autism and ADHD to the enigmatic challenges of dementia and Parkinson's, Dr. Hill breaks down how neuroplasticity isn't just a buzzword—it's your brain's ticket to a vibrant, enduring lifestyle. Tune in! 👉 Follow Claudia von Boeselager On: Instagram: https://www.instagram.com/longevityandlifestyle LinkedIn: https://www.linkedin.com/in/claudia-von-boeselager 👉 Ready to level up your life? 🚀 Discover my 10 Hacks to Improve Your Life & Longevity Playbook! Smarter, healthier, and more exciting living starts here. Grab your free copy now at longevity-and-lifestyle.com/freebie! Past guests on The Longevity & Lifestyle Podcast include Dr. David Perlmutter, Dr. Amy Killen, Sergey Young, Dr. Molly Maloof, Dr. Dale Bredesen, Shawn Wells, Dr. Dave Rabin, Dr. Kristen Willeumier, Dr. Kelly Starrett, Dr. Jack Kruse, Susan Bratton, Dr. Louise Newson, Dr. Cat Meyer, Kayla Osterhoff, Davinia Taylor, Jean Fallacara, Philipp von Holtzendorff-Fehling, Dr. Kien Vuu, Dr. Satchin Panda, Katherine Woodward Thomas, Dr. Carolina Reis, Marie Diamond, Dr. Jennifer Garrison, Nikolina Lauc, Dr. John Gray, Carrie Drinkwine, Dean Graziosi, Morri Chowaiki, Dr. Anshul Gupta, Leslie Kenny, Mohamed Massaquoi, Dr. Pamela Kryskow, Dr. Julia Mirer, Dr. Julia Jones, Dr. Austin Perlmutter, Dr. Richard Johnson, Mariko Bangerter, Kashif Khan, Matt Gallant, Andrew Lacey, Dr. Juraj Kocar, Dr. Stephanie Manson Brown, Dr. JoAnn Manson, Dr. Mohammed Enayat, Dr. Patrick Porter, Helen Reavey, Dana Frost, Lee Holden, Niall Breslin, Leighanne Champion, Dr. Louise Swartswalter, Dr. Joseph Antoun, Patrick McKeown, Dr. Alan Bauman, Max Gotzler, Dr. Marcy Cole, Dr. Dome Nischwitz, Dr. William Li, Dr. Nick Bitz, Dr. Gladys McGarey, Dr. Nichola Conlon, Dr. Sarah Myhill, Marc Brackett, Dr. Nayan Patel, Pavel Stuchlik, Alvaro Nunez, Dr. Jenny Remington-Hobbs, Jenny Pacey, Biohacker Babes, Dr. Jolene Brighten, and many more! If you enjoyed the podcast episode, please consider leaving a short review! It takes a few seconds but means the world to me to get the best guests and content for you and I love reading your reviews! Don’t forget to subscribe so you never miss an episode ➡️ @LongevityAndLifestyle Follow The Longevity & Lifestyle Podcast: ✨ Apple Podcasts: https://apple.co/3ogQDBd ✨ Spotify: https://open.spotify.com/show/77XDkcNfzZCasJXL672h01 #brainhealth #longevity #neuroplasticity
Episode Summary
I sat down with Claudia von Boeselager on the Longevity & Lifestyle podcast to talk about how you actually train a brain, what a brain map shows, and what changes across the lifespan. You can watch the original conversation. What follows is drawn from that discussion, in my own words.
What does it mean to train your brain?
You already know to check your blood lipids and back off the ice cream when the numbers drift. Your brain works the same way. You can look at the machinery inside your skull, see what is running well and what is cramped, and shape it.
The technique I work with most is neurofeedback, which trains the actual EEG, the electricity your brain produces. The starting point is a shift in perspective. Instead of receiving a diagnostic label that pulls you into a treatment-only frame, you start asking what is actually happening and whether you want to change it. That sense of agency is where the work begins.
There are about six to eight regulatory resources in the brain that are built to adjust based on how they get used. Attention. Stress response. Sleep regulation. Sensory integration and filtering. Social cueing. Speed of processing. The systems that keep things stable and resist migraine and seizure activity. These sit in the cortical areas, the top of the brain, and they manage the functional behavior of the whole system. You can train them, and you can measure them.
What is QEEG brain mapping and what does it show?
Your cortex, the bark on the surface of the brain, has columns of tissue that fire in different frequency ranges. A couple of times per second is delta. Ten times per second, 10 Hz, is alpha. These rhythms carry information, and each region tends to rest in a particular tuning mode you carry around most of the time.
A quantitative EEG, or brain map, analyzes those rhythms. We place a cap with 21 sensors plus ear clips, fill it with gel, and you sit for about 10 minutes with eyes closed and 10 minutes with eyes open. The whole thing runs 30 to 45 minutes. It does not hurt. Thick hair, braids, or dreadlocks actually make an EEG easier. A bald scalp like mine is harder, because the scalp is thicker and oilier and every pulse in the tissue shows up as artifact that mimics delta waves.
We compare your resting data to age-matched normative databases of thousands of people and build heat maps and bell curves showing where you are unusual. The goal is not to ask why you are not average. Nobody is average. The goal is to find the features that stick out and explain what each one can mean. QEEG brain mapping is not diagnostically valid on its own, because people are genuinely strange and varied. A few features come close. The classic one is a high theta-to-beta ratio over the circuits that control behavior. Theta acts like lubrication, beta like activation. Too much theta relative to beta over those control circuits is associated in the research with what we label ADHD (Monastra et al., 2001), showing up as impulsivity or inattention depending on whether the difficulty is disinhibition or low sustained attention.
We pair the resting map with a 20-minute attention test, a continuous performance task. You click for a one, withhold for a two, alternating auditory and visual trials. It is dull on purpose. Over 15 to 20 minutes you drift, you miss, you click when you should not, and that lets me pull apart stamina, consistency, and more than a dozen subcomponents of executive function rather than a single overarching label.
That same attention profile can come from several sources. Classic ADHD shows it. So do concussions, post-COVID fog, and anxiety, especially when stress and fatigue fight each other. Driving with the emergency brake on while your foot is on the floor looks a lot like ADHD, but it is usually anxiety and fatigue leaving you brittle and reactive. Reading the map alongside performance lets us model the phenomenon instead of stamping a label on it.
Where did neurofeedback come from?
The field started by accident in the 1960s. Barry Sterman at UCLA was studying how toxic rocket fuel was, exposing cats to vapor while watching for seizures. Of his cats, a subset refused to seize and stayed remarkably stable. He thought he might have found a new breed. He had not. Months earlier he had run those same cats through a different study, reinforcing them whenever they produced a particular brain rhythm. A few days of reinforcement learning had trained that rhythm, and months later those brains were seizure-resistant (Sterman et al., 1969).
Sterman then trained a research participant with medication-uncontrolled epilepsy. After about a year of audio feedback the seizure activity dropped substantially. That rhythm is sensorimotor rhythm, SMR, and it remains a core of the field (Sterman & Friar, 1972). You can read more about SMR neurofeedback and the research behind neurofeedback.
How does neurofeedback actually train a circuit?
SMR sits on the sensorimotor strip, the band of tissue running ear to ear. Just in front of the central divide is descending motor control. Just behind it is ascending sensory information. As a rough rule, the front of the brain handles internal self and doing, the back handles the outside world, the sensory and emotional tracking.
Most neurofeedback, about 95 percent of the field, is passive and transient. We measure your brain in real time. We place a couple of wires over the executive-function circuits and read three things moment to moment. Theta, the disinhibition. Alpha, sitting in neutral. Beta, voluntarily pumping the gas on those circuits. The vigilance stabilizer on the left keeps the attentional spotlight clear and on the road, and also keeps you deeply asleep at night. The circuit on the right pumps the brakes, checking and pulling back so the machine stays between the lines.
When your brain briefly moves in the trained direction, the system applauds it. The game runs faster, the music plays, the Pac-Man eats more dots. A few seconds later the brain drifts back, the game slows, and the brain notices the reward disappeared. Within the first five or ten minutes the brain starts to yoke to that information. I say brain, not mind, deliberately. My dissertation work looked at exactly this question: how does the brain know which rhythms you are rewarding? Using evoked potentials, I could see the brain create a burst or a dip in the rewarded frequency right after the feedback, within the first 10 minutes. The information loop forms before the mind participates, because you cannot feel your own brain waves.
This is implicit learning, the same way a baby does a random push-up, sees twelve feet across the room, and wants to do it again. It works on cats, who follow instructions poorly. It works on people in comas. Margaret Ayers spent much of her later career doing neurofeedback at coma bedsides, often changing the coma status. You can learn how the modern version works in this remote neurofeedback guide, and whether it holds up for ADHD and anxiety.
What does training feel like and how long does it take?
People tend to feel neurofeedback within a handful of sessions, faster than most psychological work. The pattern is consistent. You are not sure anything happened, then a session in you think you felt something, then it wears off and you assume you imagined it, then it comes back stronger. Your sleep shifts that night. Your focus or calm holds a little longer. From there you build a workout around the effects you actually noticed.
We train three times a week, the sweet spot for learning. Three times is roughly twice as effective as twice a week, while four times is only slightly better than three. With classic regulatory targets, attention, sleep, anxiety, the research suggests you can move the brain about one standard deviation on a bell curve every 20 to 25 sessions, every couple of months. We remap along the way. Most people need three to four months total, 40 to 50 sessions, to reach a durable place where the resources hold on their own. Bigger problems, large seizure disorders or significant developmental difficulty, can take six to twelve months.
Beyond the specific circuit being trained, you also get a global boost in plasticity. Single-session studies using transcranial magnetic stimulation show a reduced motor threshold after training, meaning it takes less energy to move the brain, a signature of heightened plasticity. In my coaching work this shows up sideways. I work with someone for brain fog after an injury and get a call from their physical therapist asking what changed, because the balance improved and the spasticity dropped. Teachers report kids learning better. Spouses report partners being kinder. As plasticity rises, other systems come along.
What about flow, creativity, and recovery?
There is a moment as you fall asleep where the analytical mind drops away and insight, awareness, and access surface. That hypnagogic edge is where you solve world hunger, then fall asleep and forget it. Alpha-theta neurofeedback trains you to reach that edge of consciousness and hold it for 20 to 25 minutes. Insight bubbles up, emotional access opens, and it produces a deep relaxation response. There is also evidence that alpha training raises CD4 T-cells in immune-compromised populations, almost certainly through that relaxation response. I cover related territory in biohacking flow state and the neuroscience of meditation.
The same alpha-theta work has been studied for alcohol cravings. The dry-drunk problem is real: someone sober for a decade who still cannot downshift, shaky, sleepless, on edge. After years of drinking the brain is locked in beta, heavily glutamatergic, because it stopped making the GABA that alcohol kept replacing. Train the alpha-theta resources and that GABA-driven downshift returns. Cravings fade, sleep comes back. Eugene Peniston's work reported a substantially lower one-year alcohol relapse rate when alpha-theta neurofeedback was added (Peniston & Kulkosky, 1989).
Does it work for children and older adults?
For kids there is no real low limit. QEEG normative databases run as low as six months of age. I tell people I usually start around four, because by then there is enough observable behavior and language for the child to be a participant. I have worked with toddlers freshly diagnosed as autistic, where the parents are watching the lost eye contact and the stimming and want to do something. It is harder than working with an adult. We do not run attention testing below age seven because behavior is too variable and they will not sit for it. But the feedback itself feels good, and even a nonverbal child who will only watch one movie a thousand times can train, with the movie dimming and brightening as the signal. Movies are weak feedback, because heavy social stimuli engage voluntary processing and blunt implicit learning, but they get the child to sit still while the training starts to build motor and executive control. Teenagers who do not want to be there and scroll the whole time still get changes.
The older end is more complicated, and it turns on whether the decline is truly age-related pathology. The big diseases of aging, the dementias, Parkinson's, the diabetic and cancer phenomena, are driven heavily by metabolic dysregulation. Dementia is a 20-year metabolic process, not an injury or infection, which means it can often be steered, especially early. For genuine age-related cognitive decline I point people to metabolic screening, like the work Dale Bredesen has covered on this show, and I act as a coach.
A useful signal: if someone responds to exogenous ketones, ketone esters or shakes, with a brightening and a reversal of sundowning, that suggests they still have intact neurons. In the research on Alzheimer's, neurons become insulin-resistant while sparing ketone metabolism, so ketones can support spared cognitive resources and open a window for a lower-carb, moderate-protein, higher-fat approach (Cunnane et al., 2016). Older adults absorb protein poorly, so protein often has to go higher than you would expect. Once decline is truly pathological, neurofeedback keeps plasticity high but does not have enough healthy neurons to reverse the syndrome. I suspect it can slow things like Alzheimer's, though that is extrapolation, not established.
Why isn't word-finding trouble the same as dementia?
A great deal of what people fear is dementia is not. Word-finding trouble, delayed recall for names, tip-of-the-tongue moments in your fifties and sixties: that is almost always a speed-of-processing issue, not a memory issue. Your brain stores language well and hands it up when you ask. The handoff, grabbing the word and stitching it into a sentence, is the part that slows. Look at why and you often see degraded deep sleep, the emergency brake rubbing again. It is worth addressing, the way you would address losing muscle or bone, but it does not lead to dementia.
This matters because the brain is not a bank of switches and there is no such thing as a chemical imbalance unless you are at the edge of a metabolic crisis. Parkinson's is a failure of the dopaminergic system broadly, not a low number for dopamine. The substantia nigra pars compacta produces much of the brain's dopamine, a good deal of it shipped to the frontal lobe for learning. When those basal ganglia circuits get damaged and the timing in the movement track falls apart, you get tremor. The research finds that a large fraction of dopamine-producing neurons can be lost before motor symptoms appear (Cheng et al., 2010), which tells you the brain cares about the function of the system, not the absolute level of the molecule. Dementias, likewise, are failures of mid-temporal, hippocampal, and parahippocampal tissue, not simply a low acetylcholine number. You can have a badly degraded hippocampus and perfect word-finding, because language production lives in the front left, not the mid-temporal lobe.
When is the time to act?
The brain and body stay genuinely plastic into the late sixties, sometimes later if you have been reasonably careful. After the mid-sixties, any large change in health status, body fat, or blood sugar predicts a harder road ahead. If you are heavily overweight at 70, that is not the moment to drop 50 pounds without a strong reason, because the disruption itself can trigger crises a body that age handles poorly. The move is to arrive in your mid- to late sixties with your resources dialed in. The best time to start was yesterday. The next best is today.
Where is this field heading?
I taught gerontology at UCLA for over a decade, including a course sequence on the diseases of aging. Early on those weeks were somber. By my last years teaching it, the metabolic biohacks and what became the metabolic-recode work had changed the tone entirely, because they hand people real agency. Our grandparents never looked at their triglycerides. We measure sleep, bone density, and far more.
The next step is bringing it all together. People cannot hand their supplement lists, symptom logs, and wearable data to their doctor in a form the doctor can use. As these platforms integrate, I expect intelligent agents that can run a new study or a new compound against your own QEEG, sleep logs, substance and workout history, and genetics, and tell you how it looks for you specifically, the way MTHFR status already flags certain B-vitamin choices. Personalized medicine was last decade's phrase. What replaces it looks more like a health assistant modeling all your data, so a drug-drug interaction simply does not happen because everything you take is already registered. We have more tools available than we currently use, mostly because nobody has brought them together well. That gap is exactly why people like me have work to do.
As an aside on individual variation: identical twins often, not always, show nearly the same resting brain patterns. About 90 percent of the time I look at two twin maps and have to check that they are not the same recording. The other 10 percent look like ordinary siblings.
If you take one thing from this conversation, take this. Change happens. Suffering is not as permanent as it feels. You did not always feel this way and you will not always feel this way. The regulatory features, stress, sleep, attention, speed of processing, sensory and social processing, are changeable, whether through meditation, sleep work, exercise, tailored neurofeedback, psychotherapy, or whatever comes next. You can learn more about your own machinery now than at any point in history, and just understanding how it works makes it much harder to feel overwhelmed or ashamed by it. If you want to see your own patterns, get a brain map and we will explain a little of the mystery that is you.
References
- Sterman (1972). Suppression of seizures in an epileptic following sensorimotor EEG feedback training. doi:10.1016/0013-4694(72)90028-4
- Peniston (1989). Alpha-theta brainwave training and beta-endorphin levels in alcoholics. doi:10.1111/j.1530-0277.1989.tb00325.x