In this episode, Lucas interviews Dr. Andrew Hill top peak performance coach in the US. He holds a Ph.D. in Cognitive Neuroscience from UCLA’s Department of Psychology and continues to do research on attention and cognition. He's been practicing neurofeedback since 2003 and has a wealth of knowledge in the neuroscience space. Learn more here: https://mailtrack.io/trace/link/838c5ea7c94d416ad75802af655fbd99e6c3d3a2?url=https%3A%2F%2Fpeakbraininstitute.com%2F&userId=7567458&signature=8e12eba50aafc1e2 Check Out My Website For Coaching, Recommended Products and Much More: https://www.boostyourbiology.com/ The information provided in this podcast episode is for entertainment purposes and is NOT MEDICAL ADVICE. The products presented and discussed in this podcast are explicitly only relevant to those who reside in the US. The statements and discussions held within this episode, are NOT approved by the TGA. If you have any questions about your health, contact a medical professional. This content is strictly the opinions of Lucas Aoun and is for informational and entertainment purposes only. It is not intended to provide medical advice or to take the place of medical advice or treatment from a personal physician. All viewers of this content are advised to consult with their doctors or qualified health professionals regarding specific health questions. Neither Lucas Aoun nor the publisher of this content takes responsibility for possible health consequences of any person or persons reading or following the information in this content. All consumers of this content especially taking prescription or over-the-counter medications should consult their physician before beginning any nutritional, supplement or lifestyle program.
Episode Summary
I started in neurofeedback in 2003, after a back injury ended my work doing hands-on crisis intervention at an acute psychiatric hospital. A center near me in Providence, Rhode Island was running neurofeedback for autism and ADHD, and I had years of experience with developmental populations, so I walked in and walked out with a job. Within a couple of years I was watching things I could not square with how I understood the brain to work. ADHD symptoms dropping away. Sensory issues in autism easing. Seizures going quiet in weeks. That pulled me back to UCLA for a PhD studying attention and how neurofeedback drives change in real time.
This article draws on my conversation on the Boost Your Biology Podcast with Lucas Aoun. You can watch the original conversation for the full discussion.
How does neurofeedback actually change the brain?
Most neurofeedback is passive, involuntary operant conditioning. The system measures your brain moment to moment. Say the training watches beta over the right hemisphere, an activation signal tied to whether you are engaged, alongside theta, which reads as that tissue disinhibiting, not doing its job. When the brain happens to move in the direction the protocol wants, it gets applauded by triggering a stimulus on screen. When it moves the wrong way, the stimulus stops.
The mind has no idea why the Pac-Man moved or the beep fired, because you cannot feel your own brainwaves. The brain notices anyway. It reaches for the rewarded state again, and again. Later that day or the next day, the brain starts reaching for low-theta states on its own. The mind reports it as feeling focused. Parents tell me their kid got up and cooked for themselves or did the laundry, which never happened before. Self-organization emerges as the right-sided theta gets suppressed.
I built my dissertation around what happens at the moment of reward. We ran one of the first double-blind, placebo-controlled neurofeedback studies, published in 2014 (Hill, 2014). Blinding EEG is hard, because you can see the signal move when someone coughs. The workaround stitched together stored EEG segments, scaled and blended with the person's own signal, so artifact still stopped the screen but the training parameters rewarded nothing. We showed that right after the reward fires, the brain desynchronizes and produces a burst of the trained frequency. That is the learning loop, captured in a placebo-controlled design.
The research base for neurofeedback is thinner than people expect, and there are real reasons. Blinding was only recently solved. The training is individualized and iterative, which is hard to standardize for a large trial. A gold-standard FDA study runs around five million dollars, no one owns neurofeedback, and the drug industry has spent fifty years working against it rather than funding it. So it stays a bit of a black art. For more on the evidence, see Is Neurofeedback Legitimate? A Research Overview and Does Neurofeedback Work for ADHD?.
How does brain mapping build self-awareness?
Two parts of this work change your relationship with your own physiology.
The first is the QEEG brain map. The map records the brain at rest and performance on an attention test, then compares both to a database of people your age. The point is to read the interesting shapes in performance and build a model of what you might want to change. It functions like an expanded blood panel for your brain. You learn your own physiology, and the data works for you.
Take alpha waves, the resting idle of the brain, normally around 10 Hz. If your alpha slows after your twenties, that feels like driving with the emergency brake on. People over 30 or 35 with slowed alpha get word-finding problems, tip-of-the-tongue moments, short-term memory complaints. They worry it is memory. The mechanism is speed of processing, the load time in and out of memory; individual alpha frequency tracks processing speed and memory performance in the research (Klimesch, 1999). You can map your alpha, see it running a standard deviation slow for your age, connect it to your word-finding issues, then test what shifts it. I have mapped brains under caffeine, cannabis, Adderall, modafinil, and various racetams. You can watch the speeds and amplitudes shift and compare them against the attention test you ran alongside. If piracetam speeds your alpha by a full hertz and lifts verbal fluency, now you know, instead of guessing through self-experimentation and running into side effects you did not want. More on this in Decoding Alpha Waves and Biohacking with EEG Phenotypes.
The second part is a technique called alpha-theta, which trains flow and generative access. It is the state right at sleep onset where you solve a problem or get the idea you have to write down. Alpha-theta neurofeedback trains that door to be reachable on demand. It also helps people put feelings into words, which cracks open the barrier for anyone who struggles to describe internal states. And it re-educates GABAergic tone. GABA and glutamate are the brain's main inhibitory and excitatory neurotransmitters, and they have to stay roughly balanced. Too much glutamate and you get seizures. Too much GABA and you pass out, which is why alcohol sedates you and why withdrawing it after chronic use produces seizures. A chronically wired, over-activated person who cannot fall asleep without a drink can train alpha-theta and, over a few weeks, settle into a calm state and sleep without that crutch. See Biohacking Flow State and SMR Neurofeedback.
What can neurofeedback help with?
The field started by accident. In the late 1960s, Barry Sterman exposed cats to a rocket-fuel compound and found that some of them resisted seizures. Those same cats had earlier been trained to produce more of a specific brainwave for a food reward (Sterman et al., 1969; Wyrwicka & Sterman, 1968). That brainwave is sensorimotor rhythm, the calm, laser-focused state you see in a cat watching out a window. Humans use SMR to sit still, stay asleep, suppress impulsivity, and suppress seizures. The lack of it shows up as ADHD. The calm cat and the kid who cannot sit still sit at opposite ends of the same regulatory dimension.
Beyond peak performance and attention, the two areas clients most often choose to work on are anxiety and brain fog.
Why does neurofeedback work for anxiety?
For most people, even severe presentations like PTSD or OCD, anxiety reads as natural circuits cramped up, closer to a muscle spasm than an illness. The brain map shows you which circuit.
If you cannot stop thinking about something, bite your nails, and run high-powered, the anterior cingulate, the front midline, is often producing a lot of beta. That region selects what you think about. Run it strong and you hyper-select, which becomes OCD when it gets in the way or executive drive when you can aim it. The posterior cingulate, the back midline, is the orient-and-alert region, the part that catches the frisbee and tells you to watch the road. When the brain learns the world is not safe or predictable, that region cramps up and starts scanning for danger constantly. It is like a lower back that spasmed in a car accident and stayed sore for years. You can stretch it back out. The right temporal-parietal junction handles social and sensory load, and when it is overactive the world feels too loud and a partner's chewing becomes unbearable. You can see these patterns and train them. More in Biohacking Anxiety, Neurofeedback for Anxiety, and Biohacking Fight or Flight.
What does neurofeedback do for brain fog and long COVID?
A large share of people with symptomatic COVID still report neurological or cognitive symptoms months later, based on early longitudinal data (Huang et al., 2021). About a month and a half out, many people's brains look like they have had a concussion, and they feel like it.
I have mapped thousands of people over years, often with multiple maps on the same person. When clients come back with new fog, COVID does not usually create a fresh inflammatory spot. It seems to latch onto old seeds of inflammation, old scar tissue or a prior insult, and make them flare. Alpha slows, delta rises, metabolism drops. It reads like a concussion client who got another concussion, or an apnea patient hit with mold. A double metabolic hit. For that pattern we often use hemoencephalography, training the vasculature directly, which works well for COVID brain fog in what I see. See Biohacking Brain Fog and Biohacking Plasticity.
Can a brain map show mood or depression?
You cannot predict mood from a map, though you sometimes see it. Richie Davidson's early work found a frontal asymmetry in depression, less left-frontal approach activity and more right-frontal bias, where the right frontal lobe drives toward the negative (Davidson, 1992; Henriques & Davidson, 1991). We have an evolutionary negativity bias, because missing a tiger once can end you while missing something pleasant just means there is more tomorrow. Slowed alpha and reduced processing speed often show up in depression too, but a slow processing speed produces a slow processing speed, and the map is useful for that specific bottleneck rather than for diagnosing a mood state.
Depression is a broad cortical, high-level human experience, like thoughts or memories, spread everywhere rather than living in one module. Anxiety lives in discrete resources you can see and exercise. If a map shows rumination, social or sensory overload, slowed processing, poor sleep, or attention difficulty, you can train it directly, which is the actionable advantage of EEG analysis over imaging that tells you a state but not a lever. Related reading: Where Is Love in the Brain? and The Critical Aging Window.
What does a neurofeedback program look like?
People train about three times a week, around half an hour a session. What happens in each session varies by your goals: which frequencies, where on the head, how many channels, EEG or HEG or a combination.
We start with the QEEG map, gel and cap, ten minutes eyes closed and open, plus a deliberately boring attention test that reveals how well you hold focus when nothing is exciting. Then we build a customized program, the way a good trainer would in a high-end gym. Most of our clients work remotely. We ship the mapping and training gear, and coaches work with you privately, troubleshooting setup and adjusting protocols as your sleep, stress, and attention shift.
The experience is subtle. You set up the gear, run the session, and notice whether anything feels different that day. You might feel wired, tired, slightly irritable, or notice interrupted sleep, and then it wears off, because a single push is transient. Repeat it five, ten, fifteen times and it starts to hold, because the brain is practicing something it does every day in a new way. Sleep, stress, and attention changes tend to stick reliably after a few months.
Timelines depend on the goal:
- Simple goals (ADHD, some anxiety, some sleep): 40 to 50 sessions over three to four months, often durable for most people. With ADHD, the controlled literature finds medium-to-large effects on inattention and clinically meaningful gains that hold at follow-up (Arns et al., 2009; Van Doren et al., 2019). What I see in my own coaching data is that people who start one or two standard deviations below the mean on attention testing often move well above average over a few months, but that is my observation, not a guaranteed outcome.
- Autism: about six months, since change moves slower and there is more to do.
- Active disease processes like Parkinson's or schizophrenia: longer, often ongoing.
- Peak performers: some train for years, working through anxiety, drinking, focus, and sleep, then stacking meditation, fasting, and flow-state protocols on top.
One more mechanism worth knowing: there is evidence that a single neurofeedback session can acutely modulate plasticity-related signaling for a period afterward (Ros et al., 2010). Whatever else you are doing to improve your brain may synergize with that window. For the cost side, see How Much Does Neurofeedback Cost in 2026?, Is Neurofeedback Covered by Insurance?, and Remote Neurofeedback.
What changed in my own brain?
I came to this as the most impulsive person I had ever met, well into my twenties. I did about 18 sessions and went from severe ADHD to essentially none in about six weeks. That is what made graduate school possible for me. In a separate session I trained down theta on the front midline, pushed too hard, and woke up the next day feeling like I had run a marathon. About a month later I noticed my fingernails were getting in the way of my keyboard. I had bitten my nails obsessively since around age three, and one session partitioned that behavior out of my life entirely. I have seen the same kind of thing with other people's tics and obsessions.
Those experiences shaped how I run Peak Brain. We do not chase a perfect discrete explanation for every signal, which is the trap of having to be right or risk harm. We take the trainer's approach: test, observe, adjust, stay gentle, meet you where you are, move you toward where you want to go.
Where is neurofeedback heading?
The hard part of any sophisticated coaching is the round trip, watching what happens as a change lands and steering it. We are building a quantified-self dashboard to track how sleep, stress, meditation, and diet interact with processing speed and the rest. The longer aim is to replicate skilled coaching decisions inside that interface, then mine the data for prediction, until you have a health avatar you can run tests against. Ask it what a nootropic would do, or let it warn you that your current sleep trend will put you in trouble within a week.
If you want to see your own data, the QEEG map is the place to start. It gives you a snapshot of your attention, sleep, stress, and processing speed that you can act on, and it moves a lot of what we call mental health out of the diagnostic frame and into a question of which resources are bottlenecked and how to train them out. Book a brain map, look at your alpha speed, and decide which circuit you want to work on first.
References
- Klimesch (1999). EEG alpha and theta oscillations reflect cognitive and memory performance: a review and analysis. doi:10.1016/s0165-0173(98)00056-3
- Huang (2021). The Time-Robustness Analysis of Individual Identification Based on Resting-State EEG. doi:10.3389/fnhum.2021.672946
- Davidson (1991). Left frontal hypoactivation in depression. doi:10.1037/0021-843x.100.4.535
- Arns (2019). Sustained effects of neurofeedback in ADHD: a systematic review and meta-analysis. doi:10.1007/s00787-018-1121-4
- Ros (2010). Effectiveness of Neurofeedback Training as a Treatment for Opioid-Dependent Patients. doi:10.1177/155005941004100313